Scientific Journal of Clinical Research in Dermatology

E-ISSN: 2641-4376
Pubmed NLM Abbr: Sci J Clin Res Dermatol
Journal Full Title: Scientific Journal of Clinical Research in Dermatology
Website: Click here
DOI: 10.37871 (CrossRef)
Review Process type: Double Blind
Publication fee: Click here
Plagiarism checker: iThenticate
Language: English
Ability to collect research papers: Worldwide
Organized by: SciRes Literature

License: Creative Commons License Open Access by Scientific Journal of Clinical Research in Dermatology is licensed under a Creative Commons Attribution 4.0 International License. Based on a work at SciRes Literature..

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Research Articles

Assessment of Serum Interleukin 23, 17 Level and its Relation to Disease Control in Psoriasis, Atopic Dermatitis and Lichen Planus: A Serological Study 07/02/2020

Prevalence and Epidemiological of Burns in Hebron, Palestine 03/13/2019

Acne Relapses and Maintenance Therapy: an Update on Definition and Prevention 07/02/2017

Sclerodermatous Skin: An Immunohistochemical Study 02/22/2017

The Efficacy and Safety of Touch-up Treatment with Hyaluronic Acid Filler for the Correction of Nasolabial Folds 01/03/2016

Review Articles

Adipose Tissue: Function, Adipokines, and Alterations Related with Psoriasis 10/28/2017

Articles

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International Journal of Neurological Disorders

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Wenbin Tan

University of California, United States

Biography:

• (2003-2008) PhD, Department of Neurobiology, UCLA
• (1997-2000) Master of Science in Biochemistry and Molecular Biology, Xiangya School of Medicine, Central South University, Changsha, Hunan, PR, China
• (1990-1995) Medical Degree, Xiangya School of Medicine, Central South University, Changsha, Hunan, PR, China

Research Interest:

His current research is focused on two important questions about the treatment and pathogenesis of Port Wine Stain (PWS) birthmark: Whether combination of pulsed dye laser (PDL) and topical application of anti-angiogenetic agents render better therapeutic outcomes than PDL alone for PWS treatment and The molecular mechanism(s) of PWS pathogenesis.